A part of the brain first affected by Alzheimer's disease is thinner in youth with a risk gene for the disorder, a brain imaging study has found. A thinner entorhinal cortex may render these youth more susceptible to degenerative changes and mental decline later in life. This learning and memory hub is thinner in youth with the Alzheimer's-releated ApoE4 variant of the apolipoprotein gene, perhaps lowering the threshold for adverse consequences with aging-related tissue loss.
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